Teaching mi
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The document discusses myocardial infarction (MI), also known as a heart attack, which occurs when blood flow to the heart is blocked due to a buildup of cholesterol, white blood cells, and fat in one of the coronary arteries. It causes symptoms like chest pain, shortness of breath, nausea, and potentially death. The document outlines the etiology, pathophysiology, clinical manifestations, diagnostic measures, management including pharmacological and surgical options, complications, prevention, and nursing care for a patient experiencing an MI.
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Myocardial infarction
1. Myocardial infarction, also known as a heart attack, occurs when blood flow to part of the heart is blocked, damaging heart muscle.
2. It is usually caused by a buildup of fatty plaques in the coronary arteries that supply blood to the heart. When a plaque ruptures, a blood clot forms that blocks one of the arteries.
3. Symptoms of a heart attack include chest pain or discomfort that may travel to the arm, shoulder, or jaw. Early treatment is critical to reduce damage to the heart.
Myocardial Infarction by Anita Yadav
This document discusses myocardial infarction (MI), also known as a heart attack. It provides details on:
1. The criteria for diagnosing an MI, including the rise and fall of biochemical markers and characteristic ECG changes.
2. Common risk factors for MI such as high blood cholesterol, diabetes, hypertension, smoking, male gender, and family history of heart disease.
3. The pathophysiology of an MI, including how atherosclerotic plaque can rupture and cause a thrombus to form, blocking blood flow to heart muscle.
4. How an ECG can be used to detect patterns of ischemia, injury, and infarction during an MI.
Treatment of myocardial infarction,past& present
to Know how the treatment of important disease as myocardial infarction has been developed through time
Myocardial infarction
Mr. X, a 62-year-old smoker, presented to the hospital with severe chest pain. Myocardial infarction (MI), the death of heart muscle, can be classified as ST-segment elevation MI (STEMI) or non-ST-segment elevation MI (NSTEMI) based on electrocardiogram (ECG) findings. The heart receives its blood supply from the left and right coronary arteries, which are commonly blocked by blood clots. Timely reperfusion through procedures like percutaneous coronary intervention (PCI) within 90 minutes of arrival can reduce heart muscle death, as the first hour after symptoms presents the highest likelihood of preventing cell death. Guidelines recommend fibrinolysis or PCI depending on facility capabilities and
Myocardial infarction
This document provides an overview of myocardial infarction (MI), also known as a heart attack. It defines MI as the death of heart muscle due to sustained lack of blood supply. Common causes of MI include coronary thrombosis, arteriosclerosis, infections, hypoxia, smoking, excessive fat or exercise. Symptoms include severe chest pain, sweating, nausea, shortness of breath, and abnormal heart rhythms. Treatment involves pain medications, vasodilators, anticoagulants, thrombolytic drugs, antiarrhythmics, lifestyle changes like a low-fat diet, and potentially coronary artery bypass grafting or angioplasty procedures. Nursing diagnoses for patients include pain, anxiety, impaired cardiac output, limited activity,
Acute myocardial Infarction
This document defines myocardial infarction and provides epidemiological data. It begins by defining myocardial infarction as the irreversible necrosis of heart muscle due to prolonged ischemia resulting from a blockage in a coronary artery. It then notes that over 735,000 Americans have heart attacks each year. Risk factors include increasing age, male sex, hypertension, dyslipidemia, diabetes, smoking, obesity, physical inactivity, and excessive alcohol consumption. The pathophysiology involves rupture of an atheromatous plaque leading to thrombus formation and coronary artery occlusion, causing ischemia and eventual cell death.
Cvs lab dxami-csbrp
The document discusses acute myocardial infarction (AMI), including the pathophysiology of AMI, patterns of infarction seen on ECG and microscopy, effects of reperfusion therapy, complications of AMI, and chronic ischemic heart disease. It provides images demonstrating features of AMI such as areas of necrosis, effects of reperfusion, and complications. The goal of reperfusion therapy is to limit infarct size and salvage cardiac muscle.
Acute MI Myocardial Infarction
This document provides an overview of the management of patients with coronary vascular disorders. It discusses the pathophysiology of atherosclerosis and risk factors for coronary artery disease. Types of angina, acute coronary syndrome, myocardial infarction, and diagnostic testing are described. Both non-invasive and invasive treatment options are covered, including lifestyle changes, medications, percutaneous coronary interventions like angioplasty and stenting, and coronary artery bypass graft surgery. Post-procedure complications are also mentioned.
Myocardial infarction
A 75-year-old diabetic male presented with chest pain and other symptoms of acute coronary syndrome. The most probable diagnosis is myocardial infarction. Relevant investigations include ECG, biochemical markers like CK-MB and troponin, and echocardiogram. Management involves medical therapy in emergency, possible fibrinolysis or PCI, and long term preventative treatment. Complications can include heart failure, cardiogenic shock, arrhythmias if not properly managed.
4 4-2016 myocardial infarction
The patient presented with symptoms of chest pain, shortness of breath, and sweating. ECG and blood tests showed elevated cardiac markers. This is consistent with a diagnosis of myocardial infarction (MI). MI occurs when blood flow to the heart is blocked, causing heart muscle cell death. On pathology, MI presents as areas of necrosis and inflammation. Laboratory tests for MI diagnosis include cardiac troponins, CK-MB, and myoglobin, which are more specific and sensitive than total CK or LDH. Together, the presentation and test results make the diagnosis of MI likely in this case.
Acute myocardial infarction
This document defines acute myocardial infarction and describes its predisposing factors, location, clinical presentation, management, and differential diagnosis. An AMI is caused by deficient blood supply to the heart muscle resulting in cell death. It most commonly affects the left coronary artery. Clinical features include sudden severe chest pain unrelieved by rest. Management involves oxygen, nitroglycerin, aspirin, morphine for pain relief, and transport to the hospital for further treatment.
Myocardial infarction
Myocardial Infarction- Heart ailment.
Etiology-Diagnosis-Pathophysiology-Riskfactors-Symptoms-Treatment-Medication
Myocardial infarction 16th batchPDCH
Dr. Bishnu Prasad Gautam
Dr. Sushil Shrestha
Dr. Bishnu Prasad Gautam
Dr. Sushil Shrestha
Major Risk Factors
Hypertension
Diabetes Mellitus
Hyperlipidemia
Smoking
Obesity
Family History
Stress
Sedentary Lifestyle
Pathogenesis
Atherosclerosis
Plaque Rupture
Thrombosis
Ischemia
Infarction
Morphology
Myocardial infarction
1. A myocardial infarction occurs when blood flow to the heart is blocked, damaging heart muscle.
2. It is caused most often by atherosclerosis and plaque buildup that obstruct coronary arteries.
3. Symptoms include chest pain and other signs of reduced blood supply to the heart. Diagnosis is based on symptoms, electrocardiogram changes, and blood tests showing cardiac enzyme levels.
Myocardial infarction
will help you in understanding myocardial infarction in more detail with its management and therapy with complications and with graphical knowledge you can understand it better and some laboratry test are also included in it .
myocardial infarction
This document presents information on the management of myocardial infarction presented by several students. It discusses immediate management including oxygen, analgesics, antiemetics and aspirin. Early management within the first 12 hours includes analgesics, antithrombotic therapy with antiplatelet drugs like aspirin and clopidogrel as well as anticoagulants. It also discusses anti-anginal therapy and reperfusion therapy.
Myocardial infraction sushila
This document provides information on myocardial infarction (MI), also known as a heart attack. It defines MI as necrosis of heart muscle tissue due to reduced blood flow and oxygen supply. Causes include atherosclerosis, blood clots, or spasms blocking coronary arteries. Risk factors include age, smoking, hypertension, high cholesterol, diabetes, obesity, physical inactivity, and stress. Symptoms include chest pain and shortness of breath. Diagnosis involves ECGs, blood tests of cardiac markers, and imaging tests. Treatment focuses on restoring blood flow, reducing pain and complications, and lifestyle changes to prevent future MIs.
Myocardialinfarction
Myocardial infarction, or heart attack, occurs when blood flow to the heart is blocked, damaging heart muscle. It is usually caused by a blood clot forming in one of the coronary arteries. A heart attack can lead to damage or death of heart muscle depending on how much of the heart is affected and for how long. Diagnosis involves assessing symptoms, electrocardiogram changes, and cardiac enzyme levels. Treatment focuses on restoring blood flow through clot-busting drugs or angioplasty, along with medications, monitoring, and lifestyle changes to prevent future heart attacks.
Myocardial Infarction
This document provides information about myocardial infarction (MI) or heart attack. It defines MI as death of heart muscle cells due to lack of oxygen, usually caused by a blockage in the coronary arteries. It lists risk factors for MI such as smoking, diabetes, hypertension, and family history. It describes the signs and symptoms of MI, diagnostic tests including ECG and cardiac enzymes, types of MI, and treatments including thrombolytics, angioplasty, medications, and lifestyle changes to prevent future heart attacks. The nursing management of MI focuses on reducing pain, improving perfusion, preventing complications, health education, and calling for help if symptoms worsen.
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Shock Trial
This document summarizes a study reporting on patients experiencing cardiogenic shock following acute myocardial infarction. The study analyzed 1,190 patients registered in the SHOCK Trial Registry between 1993-1997. It found that predominant left ventricular failure was the most common cause of shock (78.5%), while mechanical complications like ventricular septal rupture or tamponade caused shock in about 12% of cases. In-hospital mortality was 60% overall but higher at 87.3% for patients with ventricular septal rupture. Outcomes were better for patients receiving treatments like thrombolysis, intra-aortic balloon counterpulsation, coronary angiography, angioplasty or bypass surgery. After adjusting for treatments, early revascularization was found to
MYOCARDITIS
Myocarditis is an inflammatory disease of the myocardium that can be caused by infectious or non-infectious triggers. It is defined as inflammation of the heart muscle and can lead to dilation, thrombi formation, and degeneration of muscle fibers. Treatment involves managing symptoms through medications like ACE inhibitors, beta-blockers, and diuretics. Immunosuppressive therapy may also be used. Prognosis depends on the severity of cardiac dysfunction, with 50% resolving in 2-4 weeks but 25% developing persistent issues and 12-25% deteriorating acutely.
Shock
shock
defination: shock is defined as an acute clinical syndrome characterized by hypoperfusion and severe disfunction of vital organs.
types of shock:
hypovolaemic
cardiogenic
obstructive
distructive
neurogenic
anaphylactic
septic
pathophysiology
causes
treatment
clinical features
Meadows Syndrome as Manifested by Displaced Ischemia of the Lower Limb
Peripartum cardiomyopathy (PPMC) or Meadows syndrome is recognised as a major cause of pregnancy-related heart failure with
high morbidity and mortality. It is a condition of unknown aetiology that manifests itself as heart failure due to systolic dysfunction
of the left ventricle during the last month of pregnancy and up to 5
months after delivery.
Meadows Syndrome as Manifested by Displaced Ischemia of the Lower Limb
Peripartum cardiomyopathy (PPMC) or Meadows syndrome is recognised as a major cause of pregnancy-related heart failure with
high morbidity and mortality. It is a condition of unknown aetiology that manifests itself as heart failure due to systolic dysfunction
of the left ventricle during the last month of pregnancy and up to 5
months after delivery.
Meadows Syndrome as Manifested by Displaced Ischemia of the Lower Limb
Peripartum cardiomyopathy (PPMC) or Meadows syndrome is recognised as a major cause of pregnancy-related heart failure with
high morbidity and mortality. It is a condition of unknown aetiology that manifests itself as heart failure due to systolic dysfunction
of the left ventricle during the last month of pregnancy and up to 5
months after delivery
Meadows Syndrome as Manifested by Displaced Ischemia of the Lower Limb
Peripartum cardiomyopathy (PPMC) or Meadows syndrome is recognised as a major cause of pregnancy-related heart failure with
high morbidity and mortality. It is a condition of unknown aetiology that manifests itself as heart failure due to systolic dysfunction
of the left ventricle during the last month of pregnancy and up to 5
months after delivery.
Meadows Syndrome as Manifested by Displaced Ischemia of the Lower Limb
Peripartum cardiomyopathy (PPMC) or Meadows syndrome is recognised as a major cause of pregnancy-related heart failure with
high morbidity and mortality. It is a condition of unknown aetiology that manifests itself as heart failure due to systolic dysfunction
of the left ventricle during the last month of pregnancy and up to 5
months after delivery
Meadows Syndrome as Manifested by Displaced Ischemia of the Lower Limb
Peripartum cardiomyopathy (PPMC) or Meadows syndrome is recognised as a major cause of pregnancy-related heart failure with
high morbidity and mortality. It is a condition of unknown aetiology that manifests itself as heart failure due to systolic dysfunction
of the left ventricle during the last month of pregnancy and up to 5
months after delivery
Shock and its management
Shock and its management is summarized as follows:
1. Shock is a life-threatening condition characterized by inadequate tissue perfusion due to reduced circulating blood volume or cardiac output.
2. Shock is classified as hypovolemic, cardiogenic, septic, traumatic, neurogenic, or hypoadrenal based on etiology.
3. The pathophysiology involves reduced circulating volume, impaired tissue oxygenation, and release of inflammatory mediators, progressing from compensated to decompensated to irreversible stages if left untreated.
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A 37-year-old man presented with fever, cough, confusion and bleeding symptoms. Laboratory tests confirmed disseminated intravascular coagulation (DIC) with low platelets and clotting factors and elevated markers of fibrin breakdown. He had a lung infection with a pleural effusion and empyema requiring chest tube placement. Treatment for DIC included blood component replacement therapy and antibiotics for the infection. His coagulation parameters improved with treatment and his clinical course was uneventful.
General principles of shock
The document discusses shock, including its definition, pathophysiology, stages, classification, and management. It defines shock as a state of low tissue perfusion inadequate for normal cellular respiration. Shock is classified into six main types: hypovolemic, traumatic, cardiogenic, obstructive, septic, and neurogenic. The stages of shock include preshock, shock, and end-organ dysfunction. Management of shock involves identifying the type and treating the underlying cause while restoring tissue perfusion.
PERIPARTUM CARDIOMYOPATHY.pptx
This document discusses peripartum cardiomyopathy (PPCM), a form of heart failure that develops in the final month of pregnancy or within 5 months after delivery. It is diagnosed when no other cause of heart failure is identified. The cause is unknown but may involve viral myocarditis, an abnormal immune response during pregnancy, or hormonal and oxidative stress factors. Symptoms include dyspnea, orthopnea, cough, and edema. Treatment involves bed rest, diuretics, and immunosuppressants. About 50% of patients recover heart function within 6 months, while mortality is high for those with persistent dysfunction.
Acute myocardial-infraction
This presentation discusses acute myocardial infarction (AMI), also known as a heart attack. AMI occurs when an area of the heart muscle dies due to obstructed blood flow. The presentation covers the objectives, definition, classifications, risk factors, signs and symptoms, investigations, and treatment options for AMI. Key points include that AMI can be caused by a blockage in the coronary arteries, often due to a blood clot, and affects different areas of the heart. Risk factors include age, smoking, diabetes, hypertension, and high cholesterol. Diagnosis involves electrocardiograms, blood tests of cardiac markers, and imaging tests. Treatment involves medications like aspirin and beta blockers, thrombolysis if ST is elevated, and sometimes ang
Acute Myopericardial Syndromes
Acute myopericardial syndromes involve inflammation of the heart and surrounding tissues. This includes pericarditis (inflammation of the pericardium), myocarditis (inflammation of the myocardium), and myopericarditis (inflammation of both tissues). Viruses are a common cause. Clinical presentation varies and diagnosis is challenging, often relying on symptoms, ECG changes, cardiac imaging and biomarkers. Treatment focuses on anti-inflammatory medications and management of complications. Most cases of pericarditis are self-limiting but myocarditis carries a risk of heart failure and should warrant hospital admission.
Definitions of acute coronary syndromes.pdf
The document discusses definitions of acute coronary syndromes (ACS) and myocardial infarction (MI). It notes that while elevated troponin levels indicate MI, the underlying cause may not be ACS. The document outlines five types of MI based on pathological causes, with Type 1 being the most common ACS-related MI due to atherosclerotic plaque rupture. It emphasizes that determining the specific cause of MI is important for guiding treatment strategies.
Acute coronary syndrome (8)
The document discusses acute coronary syndrome (ACS), which occurs when a plaque in the coronary arteries ruptures and causes blockage. It describes the signs and symptoms of ACS, including chest pain. Diagnosis involves electrocardiograms, blood tests of cardiac markers, and potentially coronary angiography. The document separates ACS into categories - those with and without ST elevation. Treatment aims to break up blood clots and reopen blocked arteries using medications, angioplasty, or coronary bypass surgery.
Septic shock e-Medicine Article
This document provides an overview of septic shock, including definitions, pathophysiology, and classifications. It defines key terms like sepsis, severe sepsis, septic shock, and systemic inflammatory response syndrome (SIRS). Septic shock is a type of distributive shock caused by excessive activation of the host's immune response to infection, leading to widespread inflammation and vascular dysfunction. Mediators like cytokines and bacterial components play a role in initiating sepsis and the associated hyperdynamic circulatory response. Organ dysfunction and failure can occur as a result of impaired perfusion and coagulation abnormalities.
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Meadows Syndrome as Manifested by Displaced Ischemia of the Lower Limb
Meadows Syndrome as Manifested by Displaced Ischemia of the Lower Limb
Meadows Syndrome as Manifested by Displaced Ischemia of the Lower Limb
Meadows Syndrome as Manifested by Displaced Ischemia of the Lower Limb
Meadows Syndrome as Manifested by Displaced Ischemia of the Lower Limb
Meadows Syndrome as Manifested by Displaced Ischemia of the Lower Limb
Meadows Syndrome as Manifested by Displaced Ischemia of the Lower Limb
Meadows Syndrome as Manifested by Displaced Ischemia of the Lower Limb
Meadows Syndrome as Manifested by Displaced Ischemia of the Lower Limb
Meadows Syndrome as Manifested by Displaced Ischemia of the Lower Limb
Meadows Syndrome as Manifested by Displaced Ischemia of the Lower Limb
Meadows Syndrome as Manifested by Displaced Ischemia of the Lower Limb
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Teaching mi
- 1. MIOCARDIAL INFARCTION PRESENTED BY LIKHILA ABRAHAM 1ST YEAR MSC NURSING
- 3. MIOCARDIAL INFARCTION IS THE MEDICAL TERM FOR AN EVENT COMMONLY KNOWN AS HEART ATTACK.IT HAPPENS WHEN BLOOD STOPS FLOWING PROPERLY TO PART OF
- 4. BECAUSE ONE OF THE CORONARY ARTERY SUPPLIES BLOOD TO THE HEART DEVELOPS A BLOCKAGE DUE TO AN UNSTABILE BUILD UP OF WBC, CHOLESTEROL AND FAT. (WIKIMEDIA)
- 5. ETIOLOG Y
- 8. CHEST PAIN
- 10. DIAPHORESIS
- 13. DEATH
- 15. MANAGEMENT
- 17. COMPLICATION S
- 20. RECAPATUALIZATION DISCUSS THE NURSING PROCESS IN CARING A PATIENT ADMITTED IN EMERGENCY WARD WITH DIAGNOSIS OF MI?