Gullian barre syndrome
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Guillain-Barré syndrome is an acute polyneuropathic disorder that causes muscle weakness, loss of reflexes, and numbness in the limbs and face. It occurs when the immune system mistakenly attacks the peripheral nervous system. The symptoms can range from mild tingling to total paralysis. The disorder is usually triggered by a bacterial or viral infection. Treatment focuses on speeding recovery through measures like plasmapheresis, IVIG, respiratory therapy, and monitoring for complications like respiratory failure or paralysis. Nurses help manage symptoms, provide education, and address problems with mobility, swallowing, and sensation that arise due to the disease process.
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Gbs
Guillain-Barré syndrome is an autoimmune disorder that causes inflammation of the nerves. It is characterized by progressive weakness in the legs and arms, loss of reflexes, and ascending paralysis over 4 weeks or less. The disorder is usually preceded by a gastrointestinal or respiratory infection. Treatment involves supportive care and immunoglobulin therapy. Most patients recover, though some experience long-term weakness depending on the severity of symptoms.
GUILLAIN BARRE SYNDROME(GBS)
Guillain-Barré syndrome (GBS) is an acute immune-mediated polyneuropathy that results in demyelination and inflammation of the peripheral nervous system. It presents as rapidly progressive muscle weakness that peaks within 4 weeks. GBS is usually preceded by a bacterial or viral infection. The most common type is acute inflammatory demyelinating polyneuropathy, which involves demyelination of motor and sensory fibers. Treatment involves plasma exchange or intravenous immunoglobulin to modulate the immune response. Nursing management focuses on respiratory support, pain management, range of motion exercises, and prevention of complications.
Guillain barre syndrome
Guillain-Barré syndrome (GBS) is an acute inflammatory polyradiculoneuropathy that causes weakness and diminished reflexes. It is considered a post-infectious, immune-mediated disease that is often preceded by a respiratory or gastrointestinal infection. The typical presentation involves ascending weakness, numbness, and pain over hours to days. Treatment involves intensive care, immunomodulation like IVIG or plasma exchange, and physical/occupational rehabilitation. Prognosis is generally good, with most patients recovering over time through remyelination.
GBS Acute Polyneuropathies (GBS)
Guillain-Barré syndrome (GBS) is an acute immune-mediated polyneuropathy characterized by acute onset of peripheral and cranial nerve dysfunction and progressive muscle weakness. It is caused by an autoimmune reaction directed against peripheral nerves, often preceded by a viral infection. Clinically, GBS presents with rapidly progressive symmetric weakness, loss of tendon reflexes, and sensory symptoms like paresthesia. Electrodiagnostic studies are diagnostic in most cases and show features of demyelination like prolonged latencies and conduction block. Treatment involves supportive care and immunotherapy.
Guillain barre syndrome
Guillain-Barre Syndrome (GBS) is an acute immune-mediated polyneuropathy where peripheral nerve myelin is attacked by the immune system, resulting in muscle weakness that starts in the legs and can progress to paralysis. It is usually triggered by a prior infection and involves lymphocytic infiltration and macrophage-mediated myelin stripping. Clinical features include progressive, symmetric muscle weakness often starting proximally, absent deep tendon reflexes, sensory symptoms, and autonomic dysfunction. Diagnosis involves albuminocytologic dissociation in cerebrospinal fluid and electromyography findings. Treatment options include intravenous immunoglobulin or plasmapheresis to modify the immune response. Most patients recover, but some have persistent
Guillain barre syndrome (gbs)
Guillain-Barre Syndrome (GBS) is a post-infectious polyneuropathy that affects the peripheral nervous system. It most commonly develops following a gastrointestinal or respiratory infection. The syndrome involves demyelination of the nerves leading to progressive muscle weakness starting in the lower limbs and sometimes involving cranial nerves and autonomic dysfunction. Diagnosis is based on clinical features and supportive test findings such as elevated CSF protein and nerve conduction studies. Treatment involves supportive care and immunomodulating therapies like intravenous immunoglobulin which aid recovery beginning within a few weeks in most cases. Prognosis is generally good but respiratory or bulbar involvement can be life threatening if not properly managed.
Guillain Barre Syndrome
Guillain-Barré syndrome is an acute inflammatory polyneuropathy typically presenting with progressive ascending motor weakness. Intravenous immunoglobulin and plasma exchange are first-line treatments aimed at hastening recovery. While some vaccinations have a temporal association with Guillain-Barré syndrome, the causal relationship remains unclear and risks must be weighed against benefits of preventing severe infectious diseases. Careful supportive care including respiratory monitoring is important given potential respiratory complications.
Presentation 1 1 1
Guillain-Barré syndrome is a rare neurological disorder where the immune system attacks the peripheral nervous system, causing muscle weakness and potentially paralysis. It is triggered by respiratory or digestive infections in some cases. The exact cause is unknown but it involves antibodies attacking myelin or gangliosides in nerves outside the brain and spinal cord. Diagnosis involves tests of reflexes, nerve conduction, and spinal fluid and treatment focuses on immunotherapy or plasma exchange to control the immune system attack. Research continues to study new treatments and understand which nerve proteins are most at risk of autoimmune damage.
Guillain Barre Syndrome
Guillain-Barré syndrome is an acute inflammatory demyelinating polyneuropathy characterized by progressive muscle weakness and loss of reflexes. It is usually preceded by a gastrointestinal or respiratory infection and can cause paralysis. Treatment involves plasmapheresis or intravenous immunoglobulin to reduce antibodies, with most patients recovering fully or having minor deficits, but 5-10% having permanent disability and 3-8% dying despite intensive care.
Guillain barre syndrome
Andy Griffith contracts Guillain-Barre syndrome after getting the flu following his wife Cindi's tonsillectomy. He experiences intense pain and paralysis. Doctors are unable to diagnose him for several days until a spinal tap reveals Guillain-Barre. There is no cure, so he must wait for it to run its course while managing pain. Guillain-Barre is an autoimmune disorder where the immune system attacks the peripheral nervous system, causing inflammation of nerves and paralysis. Proper management focuses on supportive care, respiratory support, pain management, and potentially plasma exchange or IVIG treatment.
Chronic inflammatory demyelinating Polyradiculoneuropathy
Chronic Inflammatory Demyelinating Polyradiculoneuropathy (CIDP) is a rare neurological disorder characterized by progressive weakness and impaired sensory function caused by damage to the peripheral nervous system. It has an incidence rate of 1-2 per 100,000 people and predominantly affects males over 50 years old. CIDP is diagnosed based on progressive muscle weakness, reduced reflexes, nerve conduction studies showing demyelination, elevated CSF protein, and nerve biopsy with signs of demyelination. Treatment involves intravenous immunoglobulin, plasma exchange, steroids, or other immunosuppressants, with around 50% of patients experiencing improvement with therapy.
Chronic Inflammatory Demyelinating Polyneuropathy (CIDP) & Guillain Barre Syn...
This presentation provides information on Guillain-Barre syndrome (GBS) and chronic inflammatory demyelinating polyneuropathy (CIDP). It discusses their etiology, clinical presentation, diagnosis, and physiotherapy management. Both GBS and CIDP result from an autoimmune attack on peripheral nerves causing demyelination. While GBS is acute, CIDP is chronic with progressive symptoms over time. Clinical features include weakness, numbness, and sensory loss. Physiotherapy focuses on maintaining function, mobility, and recovery through various exercises depending on the patient's stage of illness. Prognosis is generally good, with around 65% of GBS patients achieving near complete recovery within a year.
Guillain Barre Syndrome
Guillain-Barre syndrome is a rare disorder where the immune system attacks the peripheral nervous system, damaging nerves and causing muscle weakness and paralysis. It has several forms but commonly results in ascending paralysis beginning in the lower extremities. It is often triggered by a bacterial or viral infection and works by demyelinating peripheral nerves. Diagnosis involves spinal fluid analysis and nerve conduction tests. Treatment focuses on supportive care like ventilation and plasma exchange or IV immunoglobulin to stop antibody damage. Nursing care monitors for complications and manages symptoms like respiratory issues, mobility, nutrition, and autonomic dysfunction.
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Guillain-Barre Syndrome
Guillain-Barré syndrome (GBS) is a disorder in which the body's immune system attacks part of the peripheral nervous system.
Guillain-Barré syndrome can affect anybody. It can strike at any age and both sexes are equally prone to the disorder. The syndrome is rare, however, afflicting only about one person in 100,000. Usually Guillain-Barré occurs a few days or weeks after the patient has had symptoms of a respiratory or gastrointestinal viral infection. Occasionally surgery will trigger the syndrome. In rare instances vaccinations may increase the risk of GBS.
After the first clinical manifestations of the disease, the symptoms can progress over the course of hours, days, or weeks. Most people reach the stage of greatest weakness within the first 2 weeks after symptoms appear, and by the third week of the illness 90 percent of all patients are at their weakest.
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Guillain - Barré syndrome
Guillain-Barré syndrome (GBS) is an acute inflammatory polyradiculoneuropathy that causes weakness and diminished reflexes. It is typically characterized by an acute monophasic illness manifesting as ascending weakness and areflexia following a gastrointestinal or respiratory infection. The classic presentation is acute inflammatory demyelinating polyradiculoneuropathy but it can also present as acute axonal forms that damage nerve axons. Diagnosis is based on progressive ascending weakness, areflexia, and supportive findings on lumbar puncture, electrodiagnostic studies, and occasionally MRI.
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Guillain-Barré Syndrome Presentation
Guillain-Barré syndrome is an acute autoimmune disease marked by inflammation of the peripheral nerves affecting the arms and legs. It involves destruction of the myelin sheath surrounding the largest sensory and motor fibers, resulting in weakness and disrupted proprioception. While the cause is unknown, it is thought to be an inappropriate immune response, possibly linked to certain vaccines. It affects about 2 per 100,000 people annually in a nondiscriminatory manner. Signs and symptoms include numbness, tingling, muscle weakness and diminished reflexes that progress distally. Most patients experience complete recovery within 2 months to 2 years through intravenous immunoglobin therapy or plasmapheresis.
Guillain Barre Syndrome
Guillain Barre Syndrome (GBS) is an acute immune-mediated inflammatory neuropathy. It is the most common cause of acute flaccid paralysis worldwide. Recent decades have seen progress in understanding the epidemiology, pathogenesis, and prognosis of GBS. The pathogenesis involves molecular mimicry between gangliosides and antigens from preceding infections like Campylobacter jejuni, leading to anti-ganglioside antibody production and complement-mediated nerve damage. Different GBS subtypes are associated with different antiganglioside antibodies and clinical courses.
Gullian barre syndrome
Guillain-Barré syndrome is an autoimmune disorder where the immune system attacks the peripheral nervous system, causing muscle weakness and possible paralysis. It often follows a viral or bacterial infection. The body's immune system mistakenly attacks and destroys the myelin sheath surrounding nerves. This damages nerve impulses and causes muscle weakness. Symptoms include progressive muscle weakness, numbness, pain, and difficulty walking or breathing. It can range from mild to life-threatening. The cause is unknown but molecular mimicry between infectious agents and nerve tissues may play a role.
Gullian barre syndrome
Guillain-Barré syndrome is an acute inflammatory demyelinating polyneuropathy characterized by progressive muscle weakness. It is an autoimmune disorder caused by the immune system attacking the peripheral nervous system. Campylobacter infection is the most common precipitant of Guillain-Barré syndrome. There are different types including acute inflammatory demyelinating polyneuropathy and Miller Fisher syndrome. Treatment involves plasmapheresis or intravenous immunoglobulin to remove antibodies and improve symptoms.
Guillain barré syndrome
Guillain-Barré syndrome is an autoimmune disorder where the immune system attacks the peripheral nervous system, causing muscle weakness and sometimes paralysis. It is usually triggered by a bacterial or viral infection. While most patients recover fully within a few months with treatment like intravenous immunoglobulin or plasmapheresis, it can in rare cases lead to respiratory failure or even death if not properly managed. The exact mechanisms are not fully understood but involve the immune system mounting an attack against nerve antigens, damaging the myelin sheath surrounding nerves.
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Gullain Barre Syndrome is an autoimmune disorder where the body's immune system attacks the peripheral nervous system, causing demyelination of nerve fibers and impairing nerve conduction. It is often preceded by a viral or bacterial infection. Clinical manifestations include ascending muscle weakness, loss of reflexes, numbness, and paralysis. Diagnosis involves physical exam findings, elevated cerebrospinal fluid protein, and electrodiagnostic testing. Treatment focuses on immunotherapy to reduce antibodies, respiratory support, and prevention of complications like infections and blood clots.
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HOMEOSTASIS
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PATHOGENESIS (REVERSIBLE & IRREVERSIBLE)
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CELL SWELLING
INTRACELLULAR ACCUMULATION
CALCIFICATION
ENZYME LEAKAGE AND CELL DEATH
ACIDOSIS & ALKALOSIS
ELECTROLYTE IMBALANCE
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Decoding Biomarker Testing and Targeted Therapy in NSCLC: The Complete Guide ...PVI, PeerView Institute for Medical Education
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Causes Of Tooth Loss
PERIODONTAL PROBLEMS ( PERIODONTITIS, GINIGIVITIS)
Systemic Causes Of Tooth Loss
1. Diabetes Mellitus
2. Female Sexual Hormones Condition
3. Hyperpituitarism
4. Hyperthyroidism
5. Primary Hyperparathyroidism
6. Osteoporosis
7. Hypophosphatasia
8. Hypophosphatemia
Causes Of Tooth Loss
CARIES/ TOOTH DECAY
Causes Of Tooth Loss
CAUSES OF TOOTH LOSS
Consequence of tooth loss
Anatomic
Loss of ridge volume both height and width
Bone loss :
mandible > maxilla
Posteriorly > anteriorly
Anatomic consequences
Broader mandibular arch with constricting maxilary arch
Attached gingiva is replaced with less keratinised oral mucosa which is more readily traumatized.
Anatomic consequences
Tipping of the adjacent teeth
Supraeruption of the teeth
Traumatic occlusion
Premature occlusal contact
Anatomic Consequences
Anatomic Consequences
Physiologic consequences
Physiologic Consequences
Decreased lip support
Decreased lower facial height
Physiologic Consequences
Physiologic consequences
Education of Patient
Diagnosis, Treatment Planning, Design, Treatment, Sequencing, and Mouth Preparation
Support for Distal Extension Denture Bases
Establishment and Verification of Occlusal Relations and Tooth Arrangements
Initial Placement Procedures
Periodic Recall
Education of Patient
Informing a patient about a health matter to
secure informed consent.
Patient education should begin at the initial
contact with the patient and should continue throughout treatment.
The dentist and the patient share responsibility for the ultimate success of a removable partial denture.
This educational procedure is especially important when the treatment plan and prognosis are discussed with the patient.
Diagnosis, Treatment Planning, Design, Treatment, Sequencing, and Mouth Preparation
Begin with thorough medical and dental histories.
The complete oral examination must include both clinical and radiographic interpretation of:
caries
the condition of existing restorations
periodontal conditions
responses of teeth (especially abutment teeth) and residual ridges to previous stress
The vitality of remaining teeth
Continued…..
Occlusal plan evaluation
Arch form
Evaluation of Occlusal relationship through mounting the diagnostic cast
The dental cast surveyor is an absolute necessity in which patients are being treated with removable partial dentures.
Mouth preparations, in the appropriate sequence, should be oriented toward the goal of
providing adequate support, stability,
retention, and
a harmonious occlusion for the partial denture.
Support for Distal Extension Denture Bases
A base made to fit the anatomic ridge form does not provide adequate support under occlusal loading.
The base may be made to fit the form of the ridge when under function.
Support for Distal Extension Denture Bases
This provides support
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Hepcidin - Regulation and its role in Iron metabolism
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Theoretically, gene therapy is the permanent solution for genetic diseases. But it has several complexities. At its current stage, it is not accessible to most people due to its huge cost. A breakthrough may come anytime and a day may come when almost every disease will have a gene therapy Gene therapy have the potential to revolutionize the practice of medicine.
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Gullian barre syndrome
- 2. Definition GB syndrome is an acute polyneuropathic disorder affecting the peripheral nervous system medical encyclopedia GB syndrome is the problem that cause muscle weakness ,loss of reflexes and numbness of your arms and face or other parts of the body Medical dictionary
- 3. GB syndrome is a disorder in which the body immune system attacks the parts of the peripheral nervous system .the first symptoms of the disorder include varying degree of weakness or tingling sensation in the legs factsheets:neurology
- 5. Etiology 1.Autoimmune reaction 2.Bactetial infection 3.Gastro and respiratory tract infection 4.Vaccines 5.Medication
- 6. Classification 1.Acute inflammatory demyelinating polyneropathy 2.Miller fisher sydrome 3.Acute motor axonal neuropathy 4.Acute motor sensory axonal neuropathy 5.Acute panautonomic neuropathy 6.Bickerstaff’s brain stem encephalitis
- 10. Oropharyngieal dysphagia ,drooling of saliva,
- 12. Facial muscle paralysis Respiratory muscle weakness Sensory impairments 1.Areflexia 2.Loss of pain/ temperature 3.Bladderdysfuction 4.Systemic changes 5,Pulse and BP variation 6,Cardiac arrhythmias
- 13. Diagnostic measures 1.History 2.Physical examination 3.Neurological examination 4.Csf examination 5.Electromyography
- 15. Management Plasmapheresis IVIG Respiratory therapy Cardiac monitoring Feeding Alpha adrenergic agents High elastic compressinon stockings Postrual drianage Chest physiotherapy
- 17. Nursing management 1.Ineffective airway clearance related to respiratory muscle weakness,/dysphagia 2.Impaired sensory perception related to disease process 3.Imbalanced nutrition related to dysphagia 4.Anxiety related to severe chest pain Deficient knowledge regarding the disease process
- 18. Thank you