This document discusses different types of immunity, including innate immunity, acquired immunity, and the differences between active and passive immunity. It provides details on natural and artificial active immunity, as well as natural and artificial passive immunity. Various methods of conferring immunity are described, such as vaccination, administration of antibodies, and herd immunity. Measurement of immunity through antibody detection and cell-mediated immunity tests are also summarized.
This document discusses active and passive immunity. It defines active immunity as the production of antibodies by one's own immune system in response to an antigen, either through natural infection or vaccination. Passive immunity is the transfer of antibodies from another individual, such as through breastfeeding. The document outlines the types of pathogens that can cause disease and the mechanisms by which they do so. It also describes the body's lines of defense, including physical barriers, phagocytic cells, inflammation, and the immune system. Within the immune system, it distinguishes between innate immunity involving non-specific responses and adaptive immunity mediated by B cells and T cells through humoral and cell-mediated immunity. The document further details the mechanisms of antibody production and action
Humoral immunity is defined as the immunity mediated by antibodies, which are secreted by B lymphocytes.
B lymphocytes secrete the antibodies into the blood and lymph
Antibodies are immune system-related proteins called immunoglobulins. Each antibody consists of four polypeptides– two heavy chains and two light chains joined to form a "Y" shaped molecule. ... This variable region, composed of 110-130 amino acids, give the antibody its specificity for binding antigen.
antibodies are a large proteins. based on electrophorosis and centrifugation anti bodies are mainly five types .these are protects on human body from various microorganisms.
This document discusses the different types of immune cells. It describes lymphocytes including B cells, T cells, and natural killer cells. It also discusses mononuclear phagocytes such as monocytes and macrophages. Granulocytic cells including neutrophils, eosinophils, and basophils are also covered. The document concludes by briefly mentioning mast cells, dendritic cells, and follicular dendritic cells.
The document discusses the immune response, including defining immune response, the consequences and types of immune response such as humoral and cell-mediated immunity. It also covers topics like antigen processing, T cell activation, the roles of cytokines and immune cells like B cells, T cells, and antibodies in the immune response.
This document defines types of hypersensitivity reactions and allergies. It describes four main types of reactions: Type 1 IgE-mediated reactions like anaphylaxis occur rapidly via histamine release. Type 2 cytotoxic reactions damage cells via IgG/IgM antibodies. Type 3 immune complex reactions involve antigen-antibody complexes activating complement and attracting inflammatory cells. Type 4 delayed hypersensitivity reactions occur via T cell activation and cytokine release over hours to days.
Type-IV hypersensitivity reactions are cell-mediated immune responses caused by cytokines from CD4+ T cells or cytotoxic CD8+ T cells. They involve the proliferation and differentiation of antigen-responsive T cells into subsets like TH1 dominated by macrophages or TH17 dominated by neutrophils. Examples include tuberculosis reactions, contact dermatitis, and autoimmune diseases like rheumatoid arthritis, multiple sclerosis, inflammatory bowel disease, psoriasis, and type 1 diabetes.
Immunoglobulins are glycoproteins called antibodies that are produced by plasma cells in response to antigens. There are five classes of immunoglobulins - IgG, IgA, IgM, IgD, and IgE - which have different structures and roles in the immune system. IgG is the most abundant antibody found in serum and body fluids. IgA is present in secretions like breast milk, saliva, and mucus to provide immunity at body surfaces. IgM is the first antibody produced during infection and activates the complement system. IgE mediates allergic reactions by binding to mast cells.
This document summarizes different aspects of disease and the body's immune response. It discusses types of diseases including those caused by pathogens, genetic disorders, toxins and environmental factors. It also describes different types of pathogens like bacteria, viruses, parasites and fungi. The document then explains how the innate immune system provides non-specific defenses through physical barriers, chemicals, inflammation and cellular responses to fight infection. It provides details on mechanisms of innate immunity like phagocytosis, acute phase proteins and fever.
This document discusses hypersensitivity and its classification. It defines hypersensitivity as an excessive immune response to an antigen that causes tissue damage. Hypersensitivity reactions are classified based on time of onset (immediate vs delayed) and mechanism (Coombs and Gell classification). Type I is an immediate allergy mediated by IgE antibodies. Type II involves antibodies binding to antigens on self cells. Type III occurs when immune complexes accumulate. Type IV is cell-mediated and causes delayed responses. The document also discusses immunoprophylaxis methods like active immunization using vaccines and passive immunization using antibodies.
Phagocytosis is the process by which phagocytes engulf and destroy foreign particles. The key steps are: 1) margination and diapedesis bring phagocytes to the infection site, 2) chemotaxis guides them to pathogens, 3) opsonins coat pathogens for attachment to phagocytes, 4) pseudopods engulf the pathogen into a phagosome, 5) the phagosome fuses with lysosomes to form a phagolysosome, where 6) lysosomal enzymes degrade the pathogen.
This document discusses various precipitation reactions and immunological techniques used to detect antigens and antibodies, including: Ouchterlony double immunodiffusion, single radial immunodiffusion, immunoelectrophoresis, and rocket electrophoresis. It explains that precipitation reactions involve two soluble reactants forming an insoluble precipitate. These techniques use diffusion and electrophoresis of antigens and antibodies in semi-solid media like agar to form visible precipitin lines, rings, or rockets, allowing detection and sometimes quantification of proteins. The techniques have various applications in medicine and clinical laboratories.
Primary and Secondary Immune Responses AhmedRiyadh17
The document discusses primary and secondary immune responses. The primary response occurs during first-time exposure to an antigen, when the immune system must learn to recognize and make antibodies against it. The secondary response occurs upon re-exposure, when immunological memory has been established and antibodies are produced more quickly. Key differences are that the secondary response has a shorter lag time, higher antibody levels, and antibodies with greater affinity compared to the primary response.
This topic covers the brief introduction of Ag and Ab in detail. Types and functions of Ig is explained in detail. Paraproteinemias is explained with simple pictures.
by Dr. N.Sivaranjani, MD
Cell mediated immunity also known as T cell immunity. it is developed by cell mediated responses and it does not involve any antibodies. Cell mediated immunity is offered by T lymphocytes and it starts developing when T cells come in contact with the antigens. In the Cell mediated immunity T cell plays one of the important role for the process of crosstalk with other immune system as well as to signal B cells to produce the antibody mediated immune response. Primary function of cell mediated response-
1) Eliminate intracellular pathogens.
2)Eliminate tumor cells.
T cells regulate proliferation and activity of other cells of the immune system : B cells, macrophages, neutrophil, etc.
introduction of adaptive immunity. classification of adaptive immunity, factor affecting it and mechanism of adaptive immunity comparison between adaptive immunity and innate immunity. characteristic of adaptive immunity . cell mediated immune responses immunoglobulins
types of immunoglobulins. functions of immunoglobulins, hypersensitivity reactions
Cell-mediated immunity involves T cells that recognize and eliminate intracellular pathogens. MHC class I presents endogenous antigens to activate CD8+ cytotoxic T cells to kill virally infected cells, while MHC class II presents exogenous antigens to CD4+ helper T cells to upregulate immune functions against bacteria. Humoral immunity involves B cells maturing into plasma cells upon antigen recognition, secreting antibodies, and leaving memory B cells to facilitate a faster response upon reexposure.
Bacteria have several virulence factors that allow them to cause infection by adhering to host cells, invading tissues, competing for nutrients, resisting the immune system, and secreting toxins. The main virulence factors discussed are adhesion through fimbriae and adhesins, invasion through enzymes, competing for iron through siderophores, resisting phagocytosis through capsules and other mechanisms, and damaging tissues through exotoxins and endotoxins. These virulence factors enable bacteria to overcome the host's defenses and cause disease.
This document discusses immunity, including innate immunity present at birth and acquired immunity developed during life through exposure to pathogens. Innate immunity provides non-specific resistance and includes mechanical barriers, secretions, cellular factors, fever and inflammation. Acquired immunity is either active (developed from natural infection or vaccination) or passive (from transfer of antibodies). Active immunity induces antibody production and cellular responses, providing durable protection. Passive immunity transfers pre-formed antibodies, offering short-term protection until the antibodies degrade.
The document discusses immunity and principles of vaccination. It covers innate immunity, which is non-specific and present from birth, and adaptive immunity, which is acquired after exposure and leads to immunological memory. Vaccination works by exposing individuals to weakened or killed pathogens to artificially increase immunity. The goals of vaccination are to provide protective immunity through memory responses and to ultimately eradicate diseases. Vaccines can provide active immunity by inducing antibody or cellular responses, or passive immunity by administering preformed antibodies.
The document discusses active and passive immunity and different types of vaccines used for immunization. It defines active immunity as the production of antibodies by one's own immune system through natural infection or artificial vaccination. Passive immunity is the transfer of antibodies from another individual. Vaccines can be live, attenuated live, inactivated/killed, toxoids, or surface antigen vaccines. They are administered through various routes and immunization schemes involve primary vaccination and booster doses to maintain immunity levels.
Passive immunity is temporary protection transferred from another individual, such as maternal antibodies. Active immunity is stimulated through natural infection or vaccination and results in the formation of memory cells that provide long-lasting protection. Vaccines work by mimicking natural infections and stimulating the immune system to develop antibodies and memory cells against targeted pathogens. Widespread vaccination benefits both individuals and communities through herd immunity.
This document discusses the principles of vaccination. It covers:
- Active and passive immunity and how they are obtained naturally or artificially through vaccination or antibody transfer.
- The components of vaccines, including antigens, antibodies, and epitopes.
- The different types of vaccines such as live attenuated, inactivated, toxoid, subunit, and polysaccharide vaccines.
- How vaccines work to produce immunity through replication or exposure to antigens without causing disease.
- The development process, safety testing, and surveillance of new vaccines.
This document discusses different types of immunity. It describes innate immunity as non-specific resistance present from birth that is not affected by prior exposure. Innate immunity involves barriers like skin and mucosa, antimicrobial proteins, and inflammation. Acquired or adaptive immunity develops during life from exposure to antigens and results in active or passive immunity. Active immunity involves the immune system making its own antibodies and cells while passive immunity involves transfer of ready-made antibodies.
Immunity can be innate or acquired. Innate immunity is non-specific and provides immediate protection against pathogens. It includes mechanical barriers like skin and mucous membranes, antimicrobial substances, and cellular responses. Acquired immunity develops from exposure to pathogens and results in long-lasting, pathogen-specific protection through antibodies and memory cells. Acquired immunity can be active, developing from natural infection or vaccination, or passive, through transfer of antibodies from mother to fetus or through antibody administration. Both active and innate immunity provide lasting protection, while passive immunity is short-term.
This document provides an overview of immunization and vaccination. It discusses the basics of active and passive immunization. It describes different types of vaccines including live attenuated, inactivated, toxoid, polysaccharide, conjugate, recombinant, and subunit vaccines. Key concepts in vaccinology like herd immunity, vaccine efficacy, and vaccine failure are explained. The principles, technique, and schedule of immunization administration are outlined. Adverse events following immunization and vaccine storage requirements are also summarized.
Vaccination: how vaccination helps to prevent diseasesLekhan Lodhi
The document discusses vaccination and immunization. It defines vaccination as stimulating protective immune responses through exposure to non-pathogenic forms of microbes. A vaccine produces specific protection against a disease by being antigenic but not pathogenic. Immunization makes a person immune or resistant to an infectious disease typically through vaccine administration. The first empirical proof of protective immunity was provided by Edward Jenner through vaccinating against smallpox using cowpox. Smallpox was possible to eradicate due to unique biological factors and the effectiveness of the smallpox vaccine. The document also discusses vaccine design, mechanisms of protection, types of vaccines including live, inactivated, toxoids, cellular fraction and recombinant vaccines, and routes and schemes of immun
This document defines and describes different types of immunity, including innate immunity, acquired immunity, local immunity, and herd immunity. It discusses the differences between innate and acquired immunity, as well as active and passive acquired immunity. Active immunity can be natural, resulting from infection, or artificial through vaccination. Passive immunity can be natural, transferred from mother to child, or artificial through administration of antibodies. Local and herd immunity are also summarized.
Immunity different types and it's detailsAtlantaArchie
Immunity divided into adaptive and innate immunity. Innate immunity is present right from birth. Adaptive immunity is that a person acquire during the course of his/ her own life.
DEFINATION OF IMMUNOLOGY,TYPES OF IMMUNOLOGY/AQUIRED/INNATE.......................................................................................................................................................................
Immunity refers to an individual's resistance to infectious microorganisms and their toxins. There are two main types of immunity: innate (native) immunity which is non-specific, and adaptive (acquired) immunity which is antigen-specific and can provide immunological memory. Innate immunity forms the first line of defense and includes physical barriers and chemical factors. Adaptive immunity is triggered by antigen exposure and results in active or passive immunity. Active immunity induces immunological memory and can be natural or artificial. Passive immunity involves transfer of ready-made antibodies and is transient. Hypersensitivity occurs when the immune response is exaggerated, and there are several types including immediate, cytotoxic, immune complex-mediated, and cell-mediated reactions
Adaptive immunity develops in response to stimuli that induce antibody production. It has two main branches: active immunity and passive immunity. Active immunity involves the immune system developing its own response through infection or vaccination, resulting in immunological memory. Passive immunity involves receiving ready-made antibodies from another source, providing immediate but temporary protection without memory. Adaptive immunity is classified as actively or passively acquired, with actively acquired immunity developing through natural exposure or vaccination, while passively acquired immunity results from maternal antibody transfer or immunoglobulin administration.
Vaccines work by exposing the immune system to harmless or inactive forms of pathogens to induce protective immunity. First generation vaccines used whole pathogens that were either live-attenuated or killed. While live vaccines induce broad immune responses, there is a risk of reversion; killed vaccines require multiple doses but are safer. Second generation subunit vaccines use isolated protein antigens to generate specific responses without risks of live vaccines. Third generation DNA vaccines contain genetic code for antigens, inducing immune responses through host cell expression without using live pathogens.
Vaccination stimulates the body's immune system for protection against infectious diseases through immunization. Vaccines introduce antigens that produce immunity without causing illness. There are two types of immunity - passive immunity acquired through antibodies and active immunity acquired through exposure and immune response. Vaccines can be live attenuated vaccines that mimic natural infection or inactivated vaccines containing non-replicating antigens. Childhood immunization in Ghana protects against diseases like measles, whooping cough, tetanus, diphtheria, polio, yellow fever, hepatitis B through vaccines administered according to an age-based schedule. Proper cold chain storage of vaccines is important to maintain their effectiveness.
The document discusses various topics related to immunization including:
- The goals of immunization are disease prevention in individuals and eventually worldwide disease eradication.
- Immunization involves administering vaccines to stimulate immunity against infectious diseases. There are active, passive, and herd types of immunization.
- Pakistan's Expanded Program on Immunization (EPI) recommends vaccines for BCG, polio, diphtheria, pertussis, tetanus, hepatitis B, Hib, and measles to be given from birth through age 15 months. Some non-EPI recommended vaccines include rotavirus, influenza, varicella, meningococcal, and pneumococcal vaccines.
The document discusses various topics related to immunization including:
- The goals of immunization are disease prevention in individuals and eventually worldwide disease eradication.
- Immunization involves administering vaccines to stimulate immunity against infectious diseases. There are active, passive, and herd types of immunization.
- Pakistan's Expanded Program on Immunization (EPI) recommends vaccines for BCG, polio, DPT, hepatitis B, Hib, pneumococcal, and measles at various ages from birth to adolescence. The program aims to provide pediatric and adult immunization against major diseases.
Active and passive immunity can be acquired naturally through infection or artificially through vaccination. Active immunity develops when the body produces its own antibodies and memory cells in response to an antigen, while passive immunity involves the transfer of ready-made antibodies from another. Vaccinations provide active immunity by exposing the immune system to an antigen without causing disease, allowing it to mount a protective memory response upon future exposure. Different types of vaccines include live attenuated, inactivated, toxoid, cellular fraction, and recombinant vaccines, which are administered through various routes to safely induce protective immunity.
The document discusses various types of immunization including their goals, definitions, and details. It covers:
- Passive and active immunity from natural and artificial sources
- Types of vaccines including live attenuated, inactivated, subunit, toxoid, and conjugate
- Details on specific vaccines like BCG, polio, diphtheria, hepatitis B, pneumococcal, rotavirus, and measles
- Schedules, efficacy, advantages, side effects and contraindications of different vaccines
- The importance of vaccination programs in reducing disease prevalence globally
The complement system is a group of proteins in the blood that helps antibodies and phagocytic cells destroy pathogens. It is activated via three pathways - classical, lectin, and alternative. Activation leads to a cascade of reactions that results in the formation of the membrane attack complex, which punches holes in the pathogen's cell membrane, killing it. Complement also aids in inflammation, phagocytosis, and immune adherence. The system is tightly regulated to prevent damage to host cells. Deficiencies can cause diseases like hereditary angioedema.
Rickettsiae are intracellular bacteria transmitted by arthropod vectors like lice, fleas and ticks. They cause diseases like typhus, spotted fever and scrub typhus. Rickettsiae grow within the cytoplasm of cells. They are Gram-negative, non-motile and cannot survive outside host cells. Diseases include epidemic typhus transmitted by lice, endemic typhus by rat fleas, and Rocky Mountain spotted fever by ticks. Scrub typhus is caused by Orientia transmitted by trombiculid mites. Laboratory diagnosis involves staining, cell culture and serology. Treatment is with doxycycline.
This document provides information on the diagnosis and treatment of tuberculosis. It discusses methods for collecting and analyzing sputum samples under microscopy and culture to identify Mycobacterium tuberculosis. It also summarizes techniques for assessing drug resistance and extra-pulmonary tuberculosis diagnosis. Treatment involves short course multidrug regimens and directly observed therapy programs.
This document discusses viral-host interactions at the cellular, individual, and community levels. At the cellular level, viruses can cause cell death, malignant transformation, cellular proliferation, or no effect. They also change the cellular architecture by producing inclusion bodies, syncytium formation from cell membrane fusion, and altering host chromosomes. Pathogenesis depends on factors like route of entry, incubation period, host immune response including antibodies and cell-mediated immunity, and non-immunological responses like interferons and temperature. Laboratory diagnosis involves microscopy, antigen demonstration, virus isolation, and serological tests. Immunoprophylaxis uses live attenuated or killed vaccines. Chemoprophylaxis targets specific viral enzymes through nucleoside analogues, protease inhibitors, and
These gram-negative bacteria - Yersinia, Pasteurella, and Francisella - are rod-shaped pathogens commonly found in rodents. Yersinia pestis causes bubonic plague via transmission from infected rats by fleas to humans. Pasteurella multocida causes hemorrhagic septicemia in animals. Francisella tularensis causes the zoonotic disease tularemia transmitted by ticks or infected meat. Laboratory identification involves culture, biochemical testing, and microscopy of these facultatively anaerobic, non-spore forming bacteria.
Viruses are obligate intracellular parasites that contain either DNA or RNA, but not both. They lack cellular organization and rely on host cells for reproduction. Viruses come in a variety of shapes and sizes, with the largest being 300nm and smallest 20nm. They infect host cells by binding to specific cell surface receptors and introducing their genetic material. Viruses then hijack the host cell's machinery to produce viral components and assemble new virus particles, which are then released to infect other cells.
- Ascaris lumbricoides, also known as the roundworm, is the largest nematode parasite that infects the human small intestine. It is distributed worldwide.
- Male roundworms are 15-30cm long while females are 20-40cm long. Females can produce up to 27 million eggs and lay up to 200,000 eggs per day that are passed in feces.
- Roundworm infection causes a range of symptoms from mild abdominal pain to intestinal obstruction, depending on whether the larvae or adult worms are causing the issues. Diagnosis involves identifying eggs or larvae in stool or gastric samples. Treatment involves anthelmintic drugs while prevention focuses on sanitation practices.
Spirochetes for undergraduate students. treponema, leptospira, borreliaRiyaz Sheriff
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This document discusses microscopy techniques. It begins with a brief history of microscopy and introduces Antonie van Leeuwenhoek, an early pioneer in microscopy. It then defines common terms used in microscopy like aberration, contrast, depth of field, immersion fluids, and numerical aperture. Specific microscopy techniques are described like dark field microscopy, which is used to view thin organisms. Phase contrast microscopy is also explained, which improves the visibility of transparent specimens without loss of resolution.
Enterococci are Gram-positive cocci that are natural inhabitants of the gastrointestinal tract. They have become important nosocomial pathogens due to their intrinsic and acquired antibiotic resistance. This study found that Enterococcus faecalis was the most common species isolated from clinical specimens in two Saudi hospitals. Many isolates showed resistance to tetracycline, ciprofloxacin, and chloramphenicol. Vancomycin resistance was observed in 3.9% of isolates, with the VanA phenotype being most common. Pulsed-field gel electrophoresis identified identical clones of E. faecalis isolated from different hospital wards, suggesting intra-hospital transmission. The high resistance rates indicate a need for improved infection control and antibiotic steward
This document provides an overview of hypersensitivity and its classification. It discusses the four main types of hypersensitivity: Type I is anaphylactic and mediated by IgE antibodies, examples include anaphylaxis and atopy. Type II involves IgG antibodies causing blood transfusion reactions, hemolytic disease of newborn, and drug-induced hemolysis. Type III hypersensitivity involves immune complex deposition that can cause localized or generalized tissue damage. Type IV is cell-mediated and causes delayed reactions like tuberculin reactions and contact dermatitis. It provides details on the mechanisms, features, and examples of each type of hypersensitivity reaction.
Methicillin resistant Staphylococcus aureus (MRSA) demonstrates resistance to methicillin and other beta-lactam antibiotics. There are two main types - hospital associated MRSA (HA-MRSA) which circulates in healthcare facilities, and community associated MRSA (CA-MRSA) which is found in the community without recent healthcare exposure. CA-MRSA strains first emerged in the 1990s and seem to have evolved from MSSA by acquiring small SCCmec cassettes. While usually associated with community infections, some studies have found that CA-MRSA strains can also cause healthcare-associated infections.
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5. Guideline: Vitamin A supplementation for infants 1-5 months of age
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Week 8 Case of Tiana-DIAGNOSIS OF FEEDING AND EATING DISORDERS CASE STUDY.pdfReliable Assignments Help
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5. Acquired active
IMMUNITY
ACQUIRED
ACTIVE
NATURAL ARTIFICIAL
Resistance developed as a result of
antigenic stimulus
Also called “Adaptive Immunity”
Active involvement of host immune
apparatus
Leads to production of antibodies /
Immunologically active cells
Takes time to set in after infection
7. Acquired active IMMUNITY
ACQUIRED
ACTIVE
NATURAL ARTIFICIAL
Once active immunity sets in
It is long lasting
One second exposure to same antigen the
immune response is quick and abundant
:SECONDARY RESPONSE
Development of humoral & cellular
immunity
Immunological memory
Active immunization is more effective and
confers better protection
May be Natural or Artificial
8. Natural active IMMUNITY
May be as a result of clinical or inapparent infection
Measles infection gives the patient life long immunity
Adults in developing countries have natural active immunity against
polio because of inapparent infections in childhood
Duration of immunity depends on the pathogen
Short term – Eg. Influenza
Long term - Eg. Measeles , chicken pox
Why common cold does not provide us immunity ???
Antigenic
variation
9. natural active IMMUNITY
Bacterial infections provide with less degree of
immunity
PREMUNITION – Seen in syphilis . The immunity to
reinfections lasts only till the original infection is
active.
Chancroid does not provide the person with any
immunity . Person may develop lesions following
reinfection even when original infection is active.
10. Artificial active immunity
• Resistance induced by vaccines
• Vaccines are preparations of live or killed
microorganisms or their products used for
immunization
• Bacterial vaccines
– Live : BCG vaccine
– Killed: Cholera vaccine
– Subunit : Typhoid Vi antigen
– Bacterial products: Tetanus toxoid
• Viral vaccines
– Live : OPV-Sabin
– Killed : IPV – Salk
– Subunit : Hepatitis B Vaccine
11. Vaccines
LIVE VACCINES
• Infection without disease
• Immunity lasts for several
years
• Booster doses MAY BE
needed
• Can be given orally or
parenterally
KILLED VACCINES
• No infective stage
• Less immunogenic
• Repeated doses needed
– Primary dose
– Booster dose
• Oral doses not effective
• Parenteral doses given with
adjuvant to increase
humoral immunity
12. Passive immunity
o No infection
o Readymade antibodies are
administered
o No latent period
o No negative phase
o Immediate protection
o Immunity lasts for short
duration till antibodies are
metabolized
o No secondary response
o Passive immunity decreases
with repetition
13. Types of passive acquired
immunity PASSIVE
NATURAL ARTIFICIAL
Resistance transferred from mother to baby
Via placenta
Breast milk – colostrum – IgA
IgM production by fetus can start from 20th week of intrauterine life
Inadequate immunity at birth
By 3 months of age – immunological independence
• < 3 months – Pediatric infections are common
Active immunization of mother provides passive immunity to infants
Eg. Tetanus toxoid during pregnancy
14. Types of passive acquired
immunity PASSIVE
NATURAL ARTIFICIALResistance transferred by
administration of antibodies
Hyper immune sera
Convalescent sera
Pooled human Ɣ globulin
Used for prophylaxis and therapy
15. Hyperimmune sera
• Anti-tetanus serum (ATS)
• Prepared from hyperimmunized horses
• Temporary protection
• Disadvantages
• Hypersensitivity
• Immune elimination
• In use
– Hyperimmune globulin of human origin
Animal origin in use
Anti-Gas gangrene sera
Anti-Botulinum sera
Antivenoms
16. Convalescent sera
• Sera of patients recovering from disease
• Contain high levels of antibody specific to the disease
• Use – Viral infections like Hepatitis A
Pooled human Ɣ globulin
• Ɣ globulin from pooled sera of healthy adults
• Has antibodies to all pathogens prevalent in the area
• Use – Rx of patients with immunodeficiencies
17. Indications for passive
immunization
• Immediate and temporary protection of a
person at risk of developing infection
• To arrest overactive active immunity
Eg. Rh incompatibility
18. Combined immunization
• Combination of active and passive methods
• Passive immunization for immediate
protection
• Tetanus
• TIG in one arm + TT in other arm
• Followed by complete schedule of tetanus
vaccination
19. Adoptive immunity
• Special type of immunization
• Injection of immunologically competent
lymphocytes TRANSFER FACTOR
• Tried in treatment of Lepromatous Leprosy
20. Local immunity
• Besredka
• Treatment of infections in a localized area
• Polio –
– Systemic immunity by IPV
• Does not prevent multiplication of virus in the gut
– This is achieved by OPV
• Influenza
– Killed vaccine brings about a humoral response
• Not enough to prevent infection
• Intra nasal live virus injection/ natural infection provides
local immunity
– IgA
21. Immunoglobulin A (IgA)
• Secretory IgA
• Produced locally by plasma cells on mucosal
surfaces / Secretory glands
• With exposure to an antigen
Specific IgA is produced
• Mucosal defense
• Handling of antigens contracted from food and
external environment
22. Herd immunity
Overall immunity in a community
Useful in control of epidemics
• When LARGE NUMBER of people in a community are
immune to a specific pathogen Herd immunity is
SATISFACTORY
• Eradication of communicable disease lies in development of
good herd immunity rather than developing individual
immunity
23. Measurement of immunity
• Practically not possible to measure accurately
• Simple method is to demonstrate the presence of a
specific antibody
– Not reliable as one pathogen will illicit immune response to
multiple antigens
• Antibody demonstration
– Agglutination
– Precipitation
– Complement fixation
– Hemagglutination inhibition
– Neutralization
– ELISA
24. • If antigenic component is identified in-vitro or
in-vivo assays can be done.
• If immunity is associated with cell mediated
immunity
– Skin tests for delayed hypersensitivity
– In-vitro tests for Cell mediated immunity
Measurement of immunity